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Although taste responses in rats and humans differ depending on the sweet stimulus, because the homology between sweet receptors in the two species is less than 70% [62], our results suggest that the diminished sweet craving effect observed in humans treated with tesofensine is most likely not due to taste aversion or impairment in sweet perception

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[2] Once administered intramuscularly, B12 is gradually released into the bloodstream and transported to the liver, where approximately half of the body's total B12 stores are held