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[125] identified five transcripts considered to be hallmarks for the general oxidative stress response regardless of the type of ROS

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This resulted in the increased Sglutathionylation of Keap1 followed by the activation of Nrf2 nuclear translocation, which resulted in the activation of p53 transcription, decrease in the intracellular Bcl2 content and elevation in the Bax content, and promotion of tumor cell apoptosis [86]

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In this study we engineered a construct that codes for glutathione reductase (GSR), an enzyme that recycles GSSG back into GSH