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Cereb Cortex 8:218226 Luttrell LM, Lefkowitz RJ (2002) The role of beta-arrestins in the termination and transduction of G-protein-coupled receptor signals
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A reduction in glycolytic progress was detected in erastin-induced ferroptosis, which might be caused by increased synthesis of ATP and oxidative phosphorylation (OXPHOS)[10, 115]
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The junctional presence of Ect2 was further demonstrated to be crucial for the maintenance of an active pool of RhoA at sites of cellcell adhesion and the promotion of junctional tension
