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A large amount of preclinical evidence has shown that lipid metabolism is active in pancreatic tumors induced by Kras, which is often manifests as the upregulation of FASN expression, subsequently enhancing de novo FA synthesis and energy metabolism and reducing lipid peroxidation levels by removing unsaturated FAs to promote self-progression [215, 216]

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Clinical and biochemical characterization of four patients with mutations in ECHS1

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