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As for other recently described donor-recipient gene polymorphism mismatches, such as the collision genotype at the LIMS1 locus (17), it has been shown that both in kidney and liver transplantation, the GSTT1 protein may function as minor histocompatibility antigen and be responsible for antibody induction leading to ABMR and poor graft function in the absence of DSAs (15, 16, 18)

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In certain scenarios, extended survival might coincide with a decline in the quality of life