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Hollander M, Wolfe DA

doi: 10.1038/s41430-018-0348-y

Understanding metabolic dysfunctions in ASD and identification of the specific markers, e.g., glutathione, vitamin E, tetrahydrobiopterin, and antioxidative enzymes such as glutathione peroxidase, paraoxonase, and superoxide dismutase, may direct ASD treatment toward specific metabolic disturbances [23,28]

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We recently reported that endothelial carnitine palmitoyltransferase 1a (Cpt1a) is reduced by hyperoxia, and that endothelial cell-specific Cpt1a knockout mice are more susceptible to developing hyperoxia-induced injury than wild type mice
