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During each visit, the subjects were seated comfortably while a catheter was introduced into a forearm vein by a qualified phlebotomist
In the presence of 100 M TMAO, PNU37883A was able to reverse the loss of contractile function, consistent with a TMAO concentration-dependent role for inducing Kir6.1 channel potentiation in dampening the contractile function in cardiomyocytes
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