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Overall the analysis of hippocampus, inferior parietal lobule (IPL), and frontal cortex regions led to the identification of a similar set of oxidatively modified proteins (by PC, 3-NT, or HNE) in AD, EAD (a transitional stage between MCI and late-stage AD), and MCI subjects, proteins that play important roles in regulating energy metabolism (e.g., Eno 1, pyruvate kinase, ATP synthase), antioxidant and detoxification systems (e.g., peroxiredoxin, MnSOD), proteasomal activity (e.g., UCH-L1), cytoskeletal integrity (e.g., DRP-2, -tubulin), protein folding (e.g., HSP70), and cellular communication and signaling (e.g., neuropolypeptide h3) (18, 19, 28, 124, 199, 304309) (TABLE 4)

Rome: FAO (2012)

Histopathology Complete histopathology was performed in 24-mo-old female [Old Con ( n = 16), and Old mAb ( n = 16)] and male mice [Old Con ( n = 15), and Old mAb ( n = 17)] following 6 mo of mAb treatment, as well as in female mice at death from the longevity study [Old Con ( n = 30), and Old mAb ( n = 20)]

highlighted that AuNP-peptide aggregates, formed through aromatic interactions, can be dissociated by thermolysin enzyme

runny nose, coughing) after strenuous exercise (Reference Nieman, Henson and Gross115)