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Intriguingly, the same pathways are increasingly implicated in the development and growth of cancer, as more and more associations and parallels between obesity, T2D and an abnormal metabolism of cancer patients become evident
References Raun, K., Hansen, B
This variant plays an opposing role by promoting oxidative stress and ferroptosis, due to its ability to maintain GPX4 in inactive oligomers [48]
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[132] found that MET was abnormally highly expressed in PD-L1-overexpressing hepatocellular carcinoma Huh-7SR cells
