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This observation may reflect an additional or synergistic anti-inflammatory effect attributed to GIP receptor agonism alongside GLP-1 receptor agonism

Subsequent studies have proved that activation of Nrf2 and inhibition of Keap1 lead to SLC7A11 upregulation, thereby promoting resistance to ferroptosis (Fan et al., 2017).The expression of SLC7A11 can also be inhibited by transcription factors such as Tumor protein p53 (p53) and Activating transcription factor 3 (ATF3)

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Furthermore, HYA considerably enhances the fecal IgA concentration in the NC/nga mouse model of atopic dermatitis (75), indicating that the protective effect of HYA on the colitis model may be attributed to the reinforcement of an epithelial barrier and an augmented S-IgA response