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However, it is increasingly clear that CRH receptors also couple to Gi (inhibiting adenylate cyclase) and Gq (stimulating phospholipase C, the processing of phosphatidylinositol 4,5-bisphosphate into inositol trisphosphate and diacylglycerol and intracellular Ca 2+ release), as well as the recruitment of beta-arrestins which counter-regulate CRH-R function via G-protein decoupling and receptor internalization/desensitization (52)

In the presence of oxygen and/or superoxide anion (O 2 ), NO forms derivatives, including peroxynitrite (ONOO ) and its protonated form peroxynitrous acid (ONOOH) (Stamler et al

Protein Cell 14 , 376381 (2023)

[DOI] [PubMed] [Google Scholar] 226.Fondell E, et al

Figures Abstract We have previously reported that acetylsalicylic acid (aspirin, ASA) induces cell cycle arrest, oxidative stress and mitochondrial dysfunction in HepG2 cells