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Mechanistic studies revealed that AS-IV inhibits JNK signaling pathway activation by downregulating phosphorylated JNK (p-JNK) levels, thereby modulating downstream apoptosis-related molecules, such as reducing the Bax/Bcl-2 ratio and suppressing caspase-3 activation, ultimately blocking the mitochondrial apoptosis pathway

Consistency and adherence to lifestyle recommendations accelerate results

Bhunia S, et al

Verdict: Limited evidence the tendon and ligament claims have a plausible biological basis, but they remain unconfirmed in human recovery studies [35][7]

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