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Description
The main phenotype of loss-of-function mutations in arGSTs is the decreased content of anthocyanins ( 1 ) 19,20,21,22,23,24,25,26,27,28 and proanthocyanidins 27 , which can be well explained by an incomplete biosynthesis due to the absence of an essential enzyme
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doi: 10.3390/nu11092128
This activity of IRE1 is independent of its role in regulating the UPR and is evolutionarily conserved
Studies in the rat
