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Rather, Grx2 overexpression cleared mutant SOD1 in the mitochondria, resulting in decreased mitochondrial fragmentation and abolishing neuron apoptosis [27]

Takaoka M, Ohkita M, Matsumura Y

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[DOI] [PubMed] [Google Scholar] Myers GJ, Davidson PW, Cox C, Shamlaye CF, Tanner MA, Marsh DO, Cernichiari E, Lapham LW, Berlin M, Clarkson TW

4.25 mg/kg GA significantly increased the number of basal melanocytes and melanoepidermal cells in shaving area of mice with hydroquinone induced vitiligo, and promote the melanin hair follicles to increase, the mechanism is to prevent oxidative stress by reducing cholinesterase (CHE) activity and MDA content and increase the expression of TYR protein