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Role of excitatory amino acid transporter2 (EAAT2) and glutamate in neurodegeneration: opportunities for developing novel therapeutics

B sung m v dng cht cho ln da khe mnh, mn mng, gim thiu cc tc ng ca lo ha

We found that A2AR deficiency in CD8 + T cells upregulated GPX4 expression (Figure 6, A and B) and A2ARi upregulated gene expression of gpx4 , which was supported by assay for transposase-accessible chromatin using sequencing (ATAC-seq) data showing increased chromatin accessibility within the vicinity of transcribed regions for gpx4 in activated CD8 + T cells upon A2ARi treatment (Figure 6C), pointing to a possible transcriptional regulation of GPX4 expression by inactivation of A2AR signaling

Lareau LF, Green RE, Bhatnagar RS, Brenner SE

Mechanistic literature demonstrates that IGF-1 supports satellite cell activation, myogenic differentiation, and regenerative remodeling, which is why IGF-1-linked markers are routinely tracked in preclinical studies involving this combination [7]