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Second, we observed deficits in endogenous remyelination in response to high rates of oligodendrocyte loss (Fig

Antioxid Redox Signal 18(2):129138

It has been safely used in a number of preclinical and clinical trials

These changes are usually temporary

In contrast, mTOR deficiency leads to hyperactivation of the TGF-Smad3 pathway and promotes Treg cell formation [155]