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To characterize other changes occurring in these mutants, we grew wildtype, sam1 /sam1 , and sam2 /sam2 strains in 15 different Phenotypic Microarray plates with different components, equal to 1440 wells, and measured for growth variations

Having delineated the core barriers that lead to the numerical paucity of tumor-specific T cells in the TME, we now turn to a contrasting layer of complexity: the numerical dominance of tumor-irrelevant bystander T cells

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Antu Kalathookunnel Antony, University of Illinois at Chicago, United States Updates Copyright 2022 Zhang, Sun, Jiang, Lu, Ding, Li, Tian and Wang