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As a result, much effort has been expended in clinical trials for various vascular diseases in the hopes that by reducing homocysteine levels, cardiovascular endpoints could be ameliorated

If done efficiently, it could explain the lack of a significant elevation in endogenous H 2 O 2 concentrations in parkin-deficient brain under steady-state conditions

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More details on neuronal signaling and synaptic mechanisms would potentially widen the model applications and level of biological detail

In this study, we evaluated the protective role of up-regulation of GSH by GGC against biomarkers of apoptosis and cell death, oxidative stress, and neuroinflammation against oligomeric A 40 in primary human astrocyte cell cultures