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Description
Regulation of mTOR by phosphatidic acid
Moreover, in response to stress, p66Shc (which functions in the intracellular pathways that convert oxidative signals into apoptosis) translocates to mitochondria to directly stimulate hydrogen peroxide generation by transferring electrons to cytochrome c
309 This reduction is achieved through partial inhibition of Complex I of the mitochondrial ETC
It has been demonstrated that hydrogen peroxide acts as an oxidant and damages the cell interrupting the signaling pathway of insulin production [30, 54, 55]
Psychological interventions have shown promise, although controlled trials in this area are limited
