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Rather than acute anaphylaxis, current data are mostly derived from studies on related disorders including allergic rhinitis, asthma, and atopic dermatitis

Since IGF-1 activates mTOR (thus, inhibits ULK1) and inhibits FoxO (thus, inhibits autophagy-related gene expression), it is reasonable to assume IGF-1 inhibits autophagy, although some conflicting results have been reported on the relative importance of mTOR and FoxO pathways in regulation of skeletal muscle autophagy

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Recently, Amaretti et

while not significant, these findings support the mechanism of action of decreasing 2,3-DPG to increase hemoglobin oxygen affinity and delay sickling