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In an in vivo model, it showed the potential to arrest the cell cycle, suppress mitosis in tumor, endorse apoptosis, down-regulate pro-survival transcription factors (survivin, Bcl-2, phospho-Akt, c-Myc, mTOR, EGFR, and VEGF), and increase p21/WAF1 activation [230]
& Rosca, E
doi: 10.1016/j.cellsig.2018.10.012
showed that intravenous injection of MSCs in mice induced neurogenesis that was due to the upregulation of Wnt/-catenin signaling [214]
Whenever possible, the source of toxin exposure should be identified and further exposure prevented
