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In research it activates VEGFR2 angiogenesis, FAK-paxillin migration and modulates NO synthesis
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The functional significance of ERK1/2 activation by BPC-157 is further supported by inhibitor studies: when the ERK pathway is pharmacologically blocked, BPC-157s pro-migratory and pro-angiogenic effects are abolished, underscoring the pathways critical role in mediating cellular activity [55]
[DOI] [PubMed] [Google Scholar] 11.Zhang Y, Proenca R, Maffei M, Barone M, Leopold L, Friedman JM
