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Growth promoting actions of IGF-II is via the IGF1R and IGF1R-INSR hybrid receptors which has preference for IGF-II actions if the A isoform of the INSR receptor expressed in the fetus and in malignantly transformed cells - pairs with the IGF1R in the hybrid (106)

provide evidence that WIF1 is a tumor suppressor and that epigenetic silencing of WIF1 accelerates osteosarcomagenesis ( 17 ).

Those prioritizing longevity and general anti-aging benefits over aggressive tissue repair may find this sufficient

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