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Conclusions In summary, our study evidenced that AT-specific B12 deficiency significantly dysregulated AT lipid metabolism and inflammation, thereby promoting dyslipidemia and worsening obesity-related complications that could underlie the development of IR and cardiometabolic risk

doi: 10.3389/fphys.2017.00240 [DOI] [PMC free article] [PubMed] [Google Scholar] [230].Holt RR, Lazarus SA, Sullards MC, et al

This catalyzes the forward reaction, a specific example of which is provided by the reactivation of peroxiredoxin-6 (Prx)

Mechanisms of redox regulation in the CNS 2.1

However, vitamin C does not reduce the incidence of PD