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Specifically, the ROS scavenger butylated hydroxyanisole delayed TNF-induced necroptosis but had no effect on necroptosis induced by direct RIPK3 oligomerization, suggesting that ROS may facilitate upstream signaling events rather than serve as an obligate execution mechanism 80

AltitudeOmics: red blood cell metabolic adaptation to high altitude hypoxia

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doi: 10.1016/j.joca.2009.03.002, PMID: [DOI] [PMC free article] [PubMed] [Google Scholar] 33.Chen J, Long F

The excess production of reactive oxygen species (ROS) could be a predominant feature not only determined the cell death but also the cell death modalities [4]