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and Lin C
Matos, J
KPV is primarily discussed for inflammation signaling and gut-related immune research, while BPC-157 is often discussed in tissue repair and healing contexts
Together, these data indicate that MV-carried OLFM4 acts as a secreted inhibitor for the Hippo signaling through a mechanism in which OLFM4 binds to extracellular domain of FAT1 to disrupt the interaction of FAT1 intracellular domain with MST1, thereby limiting MST1 activation (Fig
