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Second, in addition to vascular cells, endothelin-1 targets neurons and glial cells
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Bradley, Intraocular pressure homeostasis: maintaining balance in a high-pressure environment
The probable mechanisms by which n3-PUFAs mediate their effects in the resolution of age-related neuroinflammation are the increased synthesis of n3-PUFA-derived RvD1 and decreased n6-PUFA-derived oxylipins, displaying an anti-inflammatory profile (134, 135)
