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The above evidence explains the related mechanism of OS, HA and IR in ovulation disorders in the physiological mechanism of PCOS but does not clarify whether OS can cause PCOS ovulation disorders in other ways, such as inducing immune dysfunction or regulating gene expression

Mar, M., Morales, A., Colell, A., Garca-Ruiz, C

The GT activity was intensely reduced by 90% in response to DTNB compared to the control GT, ensuring the cysteine dependency of enzyme active sites

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Additional file 7: Table S2 (download XLSX Enrichment analysis of prospective catabolic genes from genome data of strain MBM based on KEGG pathways