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Graves Disease: Pathophysiology : An autoimmune disorder where the immune system produces thyroid-stimulating immunoglobulins (TSIs) that bind to and activate the thyroid-stimulating hormone receptor (TSHR) on thyroid follicular cells, leading to increased thyroid hormone production
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In addition, IL-32 is implicated in the maintenance of EBV latency by inactivating the activity of the Zta promoter, which is required to initiate the lytic phase [234]
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