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This process leads to the accumulation of cholesterol in the endoplasmic reticulum and increased production of 24-hydroxycholesterol (24HC)
These studies directly attribute the cytotoxicity of high-risk variants to their cation pore function
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While other mouse models with mitochondrial disturbances shift to using glucose as a fuel source, NFS1 KD results in insulin insufficiency and glucose intolerance
Ranran GAO, Zhen S, Menghui L, et al
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