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5.5 Integrated mechanistic summary and translational considerations Taken together, these mechanisms explain how organosulfur compounds (OSCs) can attenuate pathological inflammation while preserving essential host defenses when appropriately dosed: activation of the Keap1Nrf2 pathway limits oxidative stress and indirectly suppresses NF-B signaling, direct S-thiolation of upstream kinases dampens pro-inflammatory transcription, and membrane- and stress-responsive effects stabilize epithelial barriers and shape immune cell phenotypes (Dewanjee et al., 2024)

Formation of mercapturic acids

The Emerging Role of Extracellular Vesicles from Mesenchymal Stem Cells and Macrophages in Pulmonary Fibrosis: Insights into miRNA Delivery

Microbes can survive briefly on the periphery of honey, whereas pure honey quickly kills or inactivates all microbial growth due to severe osmotic conditions and acidic pH, a byproduct of honey bee salivary enzymes and microbial fermentation occurring at the hygroscopic and oxygen- rich surface

doi:10.1016/j.tem.2020.01.012