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It could be that ER oxidative capacity and the relative need for oxidized glutathione varies by cell type
Colas C, Grewer C, Otte NJ, Gameiro A, Albers T, Singh K, et al
Interleukin-1 as a therapeutic target in gout
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483 Exploiting IDH1/2 mutations with 2-HG inhibitors reverses metabolic and epigenetic dysregulation, 484 whereas combination therapies incorporating metabolic inhibitors with standard treatments block metabolic plasticity and enhance therapeutic responses
