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Consequently, the pharmacological suppression of c-Met activity has been regarded as a burgeoning approach in cancer treatment, prompting extensive global endeavors to devise more potent and targeted inhibitors
and infigratinib, afatinib, ibrutinib, and neratinib, inhibitors of various tyrosine kinases [16]
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In addition to being administered as a drug to cause oxidative stress, MTX has also been used to induce cachexia, anorexia, intestinal mucositis and digestive absorption problem in experimental animal models (Reference Boukhettala, Leblond and Claeyssens34Reference Sinno, Coquerel and Boukhettala36)
