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doi: 10.1155/2021/5598230 139 MillerKKGorceyLMcLellanBN

An animal experiment demonstrated that CD38-deficient mice exhibited significant lower levels of amyloid plaques and soluble amyloid [98]

Mechanistically, the FH / sensitivity to ferroptosis has been attributed to dysfunctional GPX4, the primary cellular defender against ferroptosis [109]

Mitochondrial dysfunction is also noted to be associated with other ASD-like neurodevelopmental disorders including Down syndrome, DiGeorge syndrome, Rett syndrome, and tuberous sclerosis complex [11,12,13,14]

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