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Bae, Y.S., Chiang, H.M., and Hickson, L., Journal of Autism and Developmental Disorders , 2015, vol

Introduction Reactive oxygen species (ROS), once perceived as toxic by-products, were known to cause oxidative damage in cells (Mittler et al., 2004

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Subsequent studies showed a role for upregulation of the pro-inflammatory mediator NFB in the GPx1 / brain following MCAO-induced injury, and found that NFB contributes, in part, to the enhanced injury in the GPx1 / mice compared to wildtype mice [108]