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We hypothesized that heterozygous C- Glud1 +/ mice exposed to stress would exhibit deficits in capacities that require intact hippocampal and mPFC function, i.e., spatial rule acquisition, cognitive flexibility and social function, whereas Glud1 heterozygosity or stress exposure alone would not elicit behavioral abnormalities

Schwann cells migrate along axons in the absence of GDNF signaling

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Extended Duration of Hyperglycemia Result in Human-like Corneal Nerve Lesions in Mice with Alloxan- and Streptozotocin-Induced Type 1 Diabetes