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[DOI] [PubMed] [Google Scholar] 132.Duncan J.W., Zhang X., Wang N., Johnson S., Harris S., Udemgba C., Ou X.M., Youdim M.B., Stockmeier C.A., Wang J.M

Similar content being viewed by others Introduction Invading pathogens or endogenous signals may induce a self-limiting immunological response called acute inflammation

System Xc-passes through a heterodimer of the light-chain subunit SLC7A11 and the heavy-chain subunit SLC3A2, and glutamate-cysteine ligase (GCL) and glutathione synthase (GSS) transfer cystine to the cell and synthesise glutathione (GSH)

However, given the limited DNA repair capacity of sperm, early intervention is crucial to prevent irreversible damage

This is the inhibition of inflammatory mediators (e.g., inducible NO synthase (iNOS), IL-6, interferon-gamma (IFN-), and TNF-) [18]
