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Targeted protein degradation: from mechanisms to clinic
Similar content being viewed by others Introduction Neuroinflammation plays a crucial role in the pathogenesis of various central nervous system (CNS) diseases, which is not only a consequence of CNS disease progression but also one of the factors driving the progression of the disease [1]
The deficit of GSH may result in a type of cell death called ferroptosis. The application of an iron chelator almost completely reverses ferroptosis which can be explained by the prevention of intralysosomal iron accumulation and leads to the suppression of induced oxidative stress permeabilization of lysosomal membranes (Homma and Fujii, 2015)
Anticancer drugs and cardiotoxicity: the role of cardiomyocyte and non-cardiomyocyte cells
The mammalian GPx family is divided into six clades according to their amino acid sequence, substrate, specificity, and subcellular localization
