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We also proved that such a defect does not result from a disorganization of the thyroid follicular structure or a lack of expression of genes involved in the T.H
[DOI] [PubMed] [Google Scholar] [100].McClung JP, Roneker CA, Mu W, Lisk DJ, Langlais P, Liu F, Lei XG, Development of insulin resistance and obesity in mice overexpressing cellular glutathione peroxidase
The growing knowledge of the gut-thyroid axis paves the way for novel treatment strategies targeting both Se levels and the microbiomes health [58]
Rimkus, C
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