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M-MDSCs primarily use pathways that do not necessarily require direct cellcell contact to inhibit effector T cell responses, achieving this through the production of nitric oxide and anti-inflammatory cytokines [24]

[PubMed] [Google Scholar] Pickart L., Vasquez-Soltero J

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As an armor-like interface and an energy buffer, it detects metabolic deviation, allocates fuels, and constrains oxidative and iron stress to stabilize axonal function

These compounds can be metabolized in the presence of placental xenobiotic-metabolizing enzymes and produce metabolites that are potentially toxic to the fetus