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Corticospinal excitability increases as the motor cortex enhances its output to compensate for deficits in spinal reflexes

There has been heightened interest in the last few years regarding genetics and ones genetic predisposition to certain acquired diseases and illnesses

In addition, supporting the critical roles of E57 TM2 and Y91 TM3 in our proposed catalytic mechanism, the mutations E57Q and Y91D have been shown to significantly reduce enzyme activity 35,49

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