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10.1021/acsinfecdis.0c00288 [DOI] [PubMed] [Google Scholar] 7.Silvagno F, Vernone A, Pescarmona GP

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They: Produce collagen Organize extracellular matrix Restore tensile structure In experimental tendon transection models, BPC-157 has been associated with: Increased fibroblast density Enhanced migration into injury site Accelerated granulation tissue formation Proposed signalling involvement includes: FAK (Focal Adhesion Kinase) activation ERK1/2 signalling Src pathway modulation FAK signalling is particularly important because it regulates: Cell adhesion Cytoskeletal reorganization Migration along collagen scaffolds Tendon strength depends not just on collagen quantity but alignment

5 Potential role of ferroptosis in RA inflammation and tissue damage 5.1 Ferroptosis products and derivatives can act as inflammatory mediators Ferroptosis is an immunogenic form of cell death, and its metabolites and derivatives can also act as inflammatory mediators to further exacerbate RA inflammatory responses