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M., Weinborn, M., Verdile, G., Sohrabi, H
The identified glutathione-related pathways provide potential therapeutic targets for alleviating oxidative stress-induced hippocampal injury in SAE, offering new insights for the clinical management of SAE-related neurological damage
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On the other hand, an excessive iodine intake, due to inappropriate supplementation or excess intake of iodine-rich food, increases the risk for hyperthyroidism/thyrotoxicosis, hypothyroidism and autoimmunity [7, 17, 24]
