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Additionally, engineered enzymes that degrade extracellular L-cystine and L-cysteine and thereby indirectly inhibit xCT, blocking intracellular GSH production and upregulating ROS, have shown promise in preclinical models 70

Supplementary Figure 5 (A, B) Cumulative data showing relative concentrations of increased metabolites in the A vs HC group

The continuous systemic inflammation generated by this vascular damage further depletes the body's already compromised antioxidant reserves, perpetuating the cycle of chronic illness and making recovery incredibly difficult without targeted biochemical intervention

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Morris, G