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Mutations in SOD1 can cause familial ALS (several pieces of evidence also show that wild-type SOD1, under conditions of cellular stress, is implicated in a significant fraction of sporadic ALS cases, which represent 90% of ALS patients.), [58] by a mechanism that is presently not understood, but not due to loss of enzymatic activity or a decrease in the conformational stability of the SOD1 protein

Currents enhanced by 0.1 M DOB returned to baseline after a 15-60-minute washing period

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In this context, physical, chemical, and microbiological approaches are applicable

Targeting increased survival may not be effective without also addressing cellular PCD resistance (93, 94)