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Jan 2016;127(1):40-49

These findings suggest that heterozygous CNTNAP2 deletions may affect genes involved in neuronal development and activity

These two monoamines, along with serotonin, have been shown to be involved in CIPN, as serotonin, norepinephrine, and dopamine reuptake inhibitors increased extracellular levels of their respective monoamines and each reversed a different combination of CIPN symptoms (Smith et al., 2013) consistent with 2+ channel inhibitor, decreased paclitaxel-induced mechanical hypersensitivity and pain aversiveness in a dose-dependent manner (Zhang et al., 2019)

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