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We then tested whether their association is affected by ROS treatment

Although THC possesses neuroprotective antioxidant properties through non-CB1R pathways [33], it also upregulates plasma inflammatory cytokines [34], increases vulnerability to ischemic stroke in young people [35], increases mitochondrial dysfunction [36, 37] and alters gene pathways associated with mitochondrial oxidative phosphorylation [38]

These events underscore the potential molecular crosstalk between both pathways, offering a fascinating opportunity to investigate novel therapeutic strategies targeting both pathways, which may enhance treatment efficacy

The clinical correlation observed between PKM2, lactate and HMGB1 levels forms a metabolic inflammatory feedback loop in the pathogenesis of sepsis

doi: 10.1111/nph.15620 88 SunY.VeerabommaS.Abdel-MageedH