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Despite ROS production may not play a crucial role in aging, it is more likely to provoke aging via oxidative damage and interaction with mitochondria [18]
Results The effect of MPTP and reserpine toxins in the presence of the BPC-157 peptide was significantly reduced
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In addition, co-culturing macrophages with sUA-exposed renal tubular cells suggest that UA amplifies inflammatory responses by triggering renal lamina propria to produce pro-inflammatory factors, instigating M1 macrophages polarization (120)
