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Statements Author contributions GS wrote and conceptualized the paper

K., Anderson, R

This effect can be completely reversed by SOD2 expression, indicating that hyperglycemia-induced OXTR suppression is due to hyperglycemia-induced consistent oxidative stress, which has been termed hyperglycemia memory ( in vivo study, which showed that OXTR expression was suppressed in many brain tissues, including the amygdala, hypothalamus and hippocampus, in prenatal diabetes exposure-induced offspring

10.1186/s40478-020-0891-3 150 LuoX

If you have a history of any of the above conditions, you may be at risk of having a zinc deficiency