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[DOI] [Google Scholar] 84.Langer S.S., Cardili R.N., Melo J.M.L., Ferriani M.P.L., Moreno A.S., Dias M.M., Bueno-Filho R., Pocente R.H.C., Roxo-Junior P., Silva J., et al

However, their dysregulation can exacerbate inflammation and tissue damage in ARDS ( Given the central role of oxidative stress and lipid peroxidation in ARDS, targeting ferroptosis represents a promising therapeutic strategy ( 2 Ferroptosis in infection and ARDS development Cell death pathways play critical roles in immune homeostasis, infection defense, and inflammation resolution

For example, simultaneously evaluating the expression, enzyme activity, and lipidomic characteristics of GPX4, SLC7A11, ACSL4, LPCAT3, FSP1, and DHODH in the same batch of patient tissues, combined with disease staging and tissue-specific differences between cartilage and synovium, can clarify the dominance of each pathway at different stages

Eur J Clin Nutr 56, Suppl 3, S14S19.CrossRefGoogle ScholarPubMed Ander, BP, Dupasquier, CM, Prociuk, MA & Pierce, GN (2003) Polyunsaturated fatty acids and their effects on cardiovascular disease

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